Two new studies recently published by two different research group suggest that Premature Ejaculation has strong genetic etiologyby PEhomepage.com Editorial Team
Researchers from
Turkey and
Finland suggest that PE may have strong genetic attributes. In the first study, published in the Asian Journal of Andrology by Ozbek et al.
the authors examined the
genotypes of the serotonin transporter gene (5-HTT) in patients with premature ejaculation ( PE ) to determine the role of genetic factors in the etiopathogenesis of PE and possibly to identify the patient subgroups. Based on the results of the study the authors suggest that the 5-HTTLPR gene plays a role in the pathophysiology of all primary PE cases. Further studies are needed to evaluate the relationship between 5-HTTLPR gene polymorphism and patient subgroup (such as primary and secondary PE) responses to selective serotonin reuptake inhibitors.
In the second study, published in the Journal of Sex & Marital Therapy by Santtila et al. the researchers from
Finland explored the balance of genetic and environmental factors on sexual dysfunctions, including Premature Ejaculation (PE) during first intercourse experience in young men. Three thousand one hundred eighty six male twins and their siblings completed questionnaires on erectile dysfunction (ED), premature ejaculation (PE) and other related issues. Twin modeling revealed a significant genetic effect for PE, but not for ED.
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