Updated May 11, 2007 at 0:32 EST.
In volume 23 of the Indian Journal of Urology published in 2007, CG McMahon reviews literature reporting how serotonin may be linked to premature ejaculation ( PE ). While the exact etiology of premature ejaculation ( PE ) is unknown, researchers are increasingly pinpointing serotonin as playing a role in the ejaculatory process. Both animal and human studies are examined and bear out the implication of serotonin and how it may be related to premature ejaculation ( PE ) .
For example, rat studies implicate serotonin in the ejaculatory processes. Since serotonin is a neurotransmitter, specific receptors must be available in order to activate or deactivate the action of the neurotransmitter. In the case of ejaculation, the 5-HT2C and 5-HT1A receptors seem to determine the speed of ejaculation. If the 5-HT2C receptors are stimulated, ejaculation takes longer, but if the 5-HT1A receptors are activated, ejaculation takes place sooner. Since these studies have been primarily conducted with male rats, more research is warranted to determine if the effects are the same in human males.
Written by the PEhomepage.com Editorial Team.
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